Interface fluid syndrome after laser in situ keratomileusis following herpetic keratouveitis.

Interface fluid syndrome after laser in situ keratomileusis following herpetic keratouveitis.
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带状疱疹性角膜葡萄膜炎后激光原位角膜磨镶术后的界面液综合征。

DOI:
10.1016/j.jcrs.2013.04.026
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发表时间:
2013
期刊:
J Cataract Refract Surg.
影响因子:
--
通讯作者:
Nishida K.
Nishida K.
中科院分区:
--
文献类型:
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作者:
Goto S;Koh S;Toda R;Soma T;Matsushita K;Maeda N;Nishida K.

文献摘要

相似文献

我们报告一个不寻常的情况下,一个28岁的男子谁开发界面液与疱疹性角膜葡萄膜炎和高眼压后,激光原位角膜磨镶术(LASIK)在左眼。使用Goldmann压平眼压计无法测量IOP;回弹眼压计显示中央角膜的IOP为30 mm Hg,上级角膜为58 mm Hg。对IOP升高的药物治疗导致积聚的界面液消退。该病例强调了LASIK术后IOP升高引起的界面液或中央角膜水肿的中央角膜IOP测量的不准确性,并显示了使用回弹眼压计测量外周角膜IOP的有效性。它还表明,葡萄膜炎可以是一个危险因素,界面流体综合征后LASIK。财务披露没有作者有经济或专有利益的任何材料或方法提到。
We report an unusual case of a 28-year-old man who developed interface fluid with herpetic keratouveitis and elevated intraocular pressure (IOP) after laser in situ keratomileusis (LASIK) in the left eye. The IOP was unmeasurable with the Goldmann applanation tonometer; rebound tonometry showed an IOP of 30 mm Hg in the central cornea and 58 mm Hg in the superior cornea. Medical treatment of the elevated IOP resulted in resolution of the accumulated interface fluid. This case highlights the inaccuracy of IOP measurements in the central cornea with interface fluid or central corneal edema caused by elevated IOP after LASIK and shows the efficacy of IOP measurements at the peripheral cornea using rebound tonometry. It also shows that uveitis can be a risk factor for interface fluid syndrome after LASIK.Financial DisclosureNo author has a financial or proprietary interest in any material or method mentioned.