The K_<ATP>-channel Opener, Nicorandil, Attenuates Monocrotaline-induced Endothelial Damage in Pulmonry Vasculature through the Enhanced Expressions of eNOS and Anti-apoptotic Factors via PI3K/Akt and MAPK Pathways
The K_<ATP>-channel Opener, Nicorandil, Attenuates Monocrotaline-induced Endothelial Damage in Pulmonry Vasculature through the Enhanced Expressions of eNOS and Anti-apoptotic Factors via PI3K/Akt and MAPK Pathways
复制标题
K_<ATP>-通道开放剂尼可地尔通过 PI3K/Akt 和 MAPK 途径增强 eNOS 和抗凋亡因子的表达，从而减轻野百合碱诱导的肺血管内皮损伤
DOI：
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发表时间：
2007
期刊：
影响因子：
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通讯作者：
Makoto Sahara
中科院分区：
文献类型：
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作者：
Sahara M;Takahashi T;Imai Y;Nakajima T;Yao A;Morita T;Hirata Y;Nagai R.;Makoto Sahara;Makoto Sahara;Makoto Sahara
