Effect of oral potassium on urinary kallikrein excretion in essential hypertension.
Effect of oral potassium on urinary kallikrein excretion in essential hypertension.
复制标题
口服钾对原发性高血压患者尿激肽释放酶排泄的影响。
DOI：
10.1007/978-1-4615-9546-5_22
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发表时间：
1989
影响因子：
--
通讯作者：
Y. Imamura
中科院分区：
文献类型：
--
作者：
E. Murakami;K. Hiwada;T. Kokubu;Y. Imamura
尿激肽释放酶和钾不仅在静态而且在动态钠状态下平行排泄。口服钾负荷刺激正常血压受试者和WHO I期原发性高血压患者尿激肽释放酶的释放。II期原发性高血压患者的尿激肽释放酶基础水平最低，口服钾负荷后尿激肽释放酶无增加。提示肾激肽释放酶-激肽系统的功能活性随高血压的发展而降低。
Urinary kallikrein and potassium were excreted in parallel in not only static but also dynamic sodium states. Oral potassium load stimulated the release of urinary kallikrein in normotensive subjects and WHO stage I essential hypertensive patients. Stage II essential hypertensive patients had the lowest value of basal level of urinary kallikrein and showed no increase in urinary kallikrein by oral potassium load. These results suggest that the functional activity of renal kallikrein-kinin system decreases with the development of essential hypertension.
显微解剖兔肾单位中活性和非活性激肽释放酶（激肽原酶活性）的定位。
影响因子：
19.6
作者：
Omata,K;Carretero,OA;Scicli,AG;Jackson,BA
通讯作者：
Jackson,BA