Kallikrein-related peptidase 6 promotes psoriasiform skin inflammation through protease-activated receptor 2-independent mechanism.
Kallikrein-related peptidase 6 promotes psoriasiform skin inflammation through protease-activated receptor 2-independent mechanism.
复制标题
激肽释放酶相关肽酶 6 通过蛋白酶激活受体 2 独立机制促进银屑病皮肤炎症。
DOI：
10.1111/exd.13204
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发表时间：
2017
期刊：
影响因子：
--
通讯作者：
Yoshida S,Ishida-Yamamoto A.
中科院分区：
文献类型：
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作者：
Iinuma S;Kishibe M;Saito N;Igawa S,Honma M;Bando Y;Yoshida S,Ishida-Yamamoto A.
本文探讨激肽释放酶相关多肽酶(KLKs)在咪喹莫特(Imiquimod，Imq)诱导的银屑病样皮炎发生发展中的作用。讨论的主题包括KLK6在促进银屑病样皮肤炎症中的作用，角质形成细胞在炎症性皮肤病中的作用，以及检测KLK6的表达和定位。
The article discusses the role of Kallikrein-related peptidases (KLKs) in the development of imiquimod (IMQ)-induced psoriasiform dermatitis. Topics discussed include role of Klk6 in promoting psoriasiform skin inflammation, role of keratinocytes in inflammatory skin diseases and examining the expression and localization of Klk6.