A permissive role of pertussis toxin substrate G-protein in P2-purinergic stimulation of phosphoinositide turnover and arachidonate release in FRTL-5 thyroid cells. Cooperative mechanism of signal transduction systems.
A permissive role of pertussis toxin substrate G-protein in P2-purinergic stimulation of phosphoinositide turnover and arachidonate release in FRTL-5 thyroid cells. Cooperative mechanism of signal transduction systems.
复制标题
百日咳毒素底物 G 蛋白在 P2 嘌呤能刺激 FRTL-5 甲状腺细胞中磷酸肌醇周转和花生四烯酸释放中的许可作用。
DOI：
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复制
发表时间：
1989
影响因子：
4.8
通讯作者：
Y. Kondo
中科院分区：
文献类型：
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作者：
F. Okajima;K. Sato;M. Nazarea;K. Sho;Y. Kondo
