Nutrition Education to Reduce Metabolic Dysfunction for Spinal Cord Injury: A Module-Based Nutrition Education Guide for Healthcare Providers and Consumers.

Nutrition Education to Reduce Metabolic Dysfunction for Spinal Cord Injury: A Module-Based Nutrition Education Guide for Healthcare Providers and Consumers.
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营养教育减少脊髓损伤代谢功能障碍:基于模块的医疗保健提供者和消费者的营养教育指南。

DOI:
10.3390/jpm12122029
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发表时间:
2022-12-07
影响因子:
--
通讯作者:
Gater, David R.
Gater, David R.
中科院分区:
医学4区
文献类型:
--
作者:
Sneij, Alicia;Farkas, Gary J.;Carino Mason, Marisa Renee;Gater, David R.

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脊髓损伤(SCI)导致神经源性肥胖和代谢功能障碍的高患病率。神经源性肥胖和代谢功能障碍的风险增加主要是由于能量平衡的丧失,因为SCI后能量消耗显著减少。因此,过量的能量摄入(正能量平衡)导致脂肪组织快速积累,导致神经源性肥胖、全身炎症和代谢功能障碍。本文的目的是回顾现有的文献营养,膳食摄入量,营养教育的人与SCI,因为它涉及到代谢功能障碍。这项检讨将根据权威指引,强调脊髓损伤患者的膳食摄入量不足,以及有需要向医护人员和消费者提供营养教育。营养教育主题以模块为基础的格式与支持文献。作者强调了由低能量、营养密集、抗炎食物组成的饮食的作用,这些食物与美国人MyPlate饮食指南一致,可以有效地实现能量平衡,降低SCI患者神经源性肥胖和代谢功能障碍的风险。
Spinal cord injury (SCI) results in a high prevalence of neurogenic obesity and metabolic dysfunction. The increased risk for neurogenic obesity and metabolic dysfunction is mainly due to the loss of energy balance because of significantly reduced energy expenditure following SCI. Consequently, excessive energy intake (positive energy balance) leads to adipose tissue accumulation at a rapid rate, resulting in neurogenic obesity, systemic inflammation, and metabolic dysfunction. The purpose of this article is to review the existing literature on nutrition, dietary intake, and nutrition education in persons with SCI as it relates to metabolic dysfunction. The review will highlight the poor dietary intakes of persons with SCI according to authoritative guidelines and the need for nutrition education for health care professionals and consumers. Nutrition education topics are presented in a module-based format with supporting literature. The authors emphasize the role of a diet consisting of low-energy, nutrient-dense, anti-inflammatory foods consistent with the Dietary Guidelines for Americans’ MyPlate to effectively achieve energy balance and reduce the risk for neurogenic obesity and metabolic dysfunction in individuals with SCI.
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