[Intralymphatic histiocytosis and cancer of the colon].

[Intralymphatic histiocytosis and cancer of the colon].
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[淋巴内组织细胞增多症和结肠癌]。

DOI:
10.1016/s1578-2190(10)70625-9
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发表时间:
2010
影响因子:
3.2
通讯作者:
C. Guillén
C. Guillén
中科院分区:
--
文献类型:
--
作者:
B. Echeverría‐García;R. Botella;C. Requena;C. Guillén

文献摘要

参考文献

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相似文献

淋巴管内组织细胞增生症是一种无特异性临床特征,但具有特征性组织病理学和免疫组化表现的疾病。它表现为边界不清的斑块或类似网状青斑的病变,通常位于四肢。组织学上,在网状真皮中观察到含有单核细胞的扩张血管。在过去,大多数病例被认为是反应性血管内皮瘤病或血管内淋巴瘤,但更特异性的免疫组织化学标记的发展表明,这些是含有组织细胞的扩张的淋巴管,因此被认为是一个单独的条件。我们提出的情况下,一名男子与过去的历史左腋窝淋巴结清扫淋巴结核,白细胞破碎性血管炎,结肠癌。患者在左胸区域出现动脉粥样硬化斑块。组织学及免疫组化染色显示真皮网状层扩张的淋巴管内有组织细胞,但无肿瘤细胞。我们提出了一个新的情况下,淋巴组织细胞增多症的患者与自身免疫性和肿瘤性疾病。
Intralymphatic histiocytosis is a condition with nonspecific clinical features but with characteristic histopathological and immunohistochemical findings. It presents as a poorly demarcated erythematous plaque or a lesion similar to livedo reticularis, usually located on the limbs. Histologically, dilated vessels containing mononuclear cells are observed in the reticular dermis. In the past, the majority of these cases were thought to be cases of reactive angioendotheliomatosis or intravascular lymphoma, but the development of more specific immunohistochemical markers showed that these were dilated lymph vessels containing histiocytes, and so were considered as a separate condition. We present the case of a man with a past history of left axillary lymphadenectomy for lymphatic tuberculosis, leukocytoclastic vasculitis, and cancer of the colon. The patient developed an erythematous plaque in the left pectoral region. Histology and immunohistochemical stains of the lesion revealed histiocytes within dilated lymph vessels in the reticular dermis, but no neoplastic cells. We present a new case of intralymphatic histiocytosis in a patient with autoimmune and neoplastic diseases.
DOI: 10.1097/dad.0b013e3181986cc2
发表时间: 2009-04-01
影响因子: 1.1
作者:
Requena, Luis;El-Shabrawi-Caelen, Laila;Kutzner, Heinz
通讯作者: Kutzner, Heinz
与类风湿性关节炎相关的血管内组织细胞增多症:伴有淋巴管内皮增殖的报告
DOI: --
发表时间: 2005
期刊: Br J Dermatol 152(6)
影响因子: --
作者:
Okazaki A;Asada H;Niizeki H;Nonomura A;Mjyagawa S
通讯作者: Mjyagawa S