A central-peripheral asymmetry in masked priming

A central-peripheral asymmetry in masked priming
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DOI:
10.3758/bf03212139
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发表时间:
2000-10-01
期刊:
PERCEPTION & PSYCHOPHYSICS
影响因子:
--
通讯作者:
Eimer, M
Eimer, M
中科院分区:
其他
文献类型:
--
作者:
Schlaghecken, F;Eimer, M

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在目标之前呈现的掩蔽启动在不相容试验中产生行为益处(启动和目标映射到相反的反应),当它们出现在固定时,但在相容试验中的行为益处(其中启动和目标映射到相同的反应),当它们出现在外围时。在实验1中,这种中央-外周不对称(CPA)的时间过程进行了研究。对于中央启动,在短刺激发作期(SOA)的兼容试验的好处变成了不兼容试验的好处,在较长的SOA。对于外周预充,短SOA的兼容性试验益处随着SOA的延长而增加。实验2表明,这些影响也发生时,启动和目标是物理上不同的,排除了解释的刺激材料的感知特性。在实验3和实验4中,研究了CPA是否与视觉空间注意力和/或视网膜偏心率本身有关的问题。结果表明,CPA是独立的注意力因素,但强烈相关的视网膜的生理不均匀性。有人认为,中央和周边启动触发初始的运动激活,这是抑制只有当启动在视网膜的位置足够高的感知灵敏度。结果进行了讨论的激活阈值模型。
Masked primes presented prior to a target result in behavioral benefits on incompatible trials tin which the prime and the target are mapped onto opposite responses) when they appear at fixation, but in behavioral benefits on compatible trials (in which the prime and the target are mapped onto the same response) when appearing peripherally. In Experiment 1, the time course of this central-peripheral asymmetry (CPA) was investigated. For central primes, compatible-trial benefits at short stimulus onset asynchronies (SOAs) turned into incompatible-trial benefits at longer SOAs. For peripheral primes, compatible-trial benefits at short SOAs increased in size with longer SOAs. Experiment 2 showed that these effects also occur when primes and targets are physically dissimilar, ruling out an interpretation in terms of the perceptual properties of the stimulus material. In Experiments 3 and 4, the question was investigated as to whether the CPA is related to visual-spatial attention and/or retinal eccentricity per se. The results indicate that the CPA is independent of attentional factors but strongly related to the physiological inhomogeneity of the retina It is argued that central and peripheral primes trigger an initial motor activation, which is inhibited only if primes are presented at retinal locations of sufficiently high perceptual sensitivity. The results are discussed in terms of an activation threshold model.