Excitotoxic lesions of the gustatory thalamus spare simultaneous contrast effects but eliminate anticipatory negative contrast: evidence against a memory deficit.
Excitotoxic lesions of the gustatory thalamus spare simultaneous contrast effects but eliminate anticipatory negative contrast: evidence against a memory deficit.
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味觉丘脑的兴奋性毒性损伤可以避免同时的对比效应，但消除预期的负对比：反对记忆缺陷的证据。
DOI：
10.1037/0735-7044.118.2.365
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发表时间：
2004
期刊：
影响因子：
--
通讯作者：
Trifunovic,Radmila
中科院分区：
文献类型：
--
作者：
Reilly,Steve;Bornovalova,Marina;Trifunovic,Radmila
使用完善的对比程序和相同的味觉刺激（0.15%糖精和1.0 M蔗糖），作者在2个实验中检验了味觉丘脑病变破坏味觉记忆的假设。在实验1中，无论溶液间间隔的持续时间（0 s，30 s，1 min，2 min，4 min，8 min），丘脑病变对同时对比效应的表达没有影响。在实验2中，丘脑病变取消了预期的负对比度在0-s intersolution间隔。这些结果不支持实验假设。相反，这些数据似乎最好解释为病变引起的比较机制的破坏，负责预期的负对比。通过这一分析，不同的比较机制下的同时和预期的对比效果。
Using consummatory contrast procedures and the same taste stimuli (0.15% saccharin and 1.0 M sucrose), the authors tested the hypothesis that lesions of the gustatory thalamus disrupt gustatory memory in 2 experiments. In Experiment 1, irrespective of the duration of the intersolution interval (0 s, 30 s, 1 min, 2 min, 4 min, 8 min), thalamic lesions had no influence on the expression of simultaneous contrast effects. In Experiment 2, thalamic lesions abolished anticipatory negative contrast at the 0-s intersolution interval. These results provide no support for the experimental hypothesis. Rather, the data seem best interpreted as a lesion-induced disruption of the comparison mechanism responsible for anticipatory negative contrast. By this analysis, different comparison mechanisms underlie simultaneous and anticipatory contrast effects.