Priming across modalities and priming across category levels: extending the domain of preserved function in amnesia.
Priming across modalities and priming across category levels: extending the domain of preserved function in amnesia.
复制标题
跨模式启动和跨类别级别启动：扩展健忘症中保留功能的范围。
DOI：
10.1037//0278-7393.11.2.386
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发表时间：
1985
期刊：
影响因子：
--
通讯作者：
Squire,LR
中科院分区：
文献类型：
--
作者：
Graf,P;Shimamura,AP;Squire,LR
记忆缺失被认为反映了对特定大脑系统的损伤的影响，这些大脑系统是进行加工、巩固和有意识回忆所必需的。因此，作者认为对遗忘症的研究是建立功能分离和理解记忆功能正常组织的有用方法。本研究包括8例39-74岁的酒精性Korsakoff综合征患者，3例45-56岁的酒精性失忆症患者，18例37-63岁的酒精性男性（对照组）和16例25-61岁的正常对照组。在实验I中，通过单词完成测试测量，所有组都表现出显着的启动效应，在一种模式比跨模式。尽管记忆力严重受损，但在内部和跨模式中，记忆障碍者表现出正常的启动效应。实验二中，所有被试在被试被提供类别标签时，均表现出显著的、等效的类别样例启动，被试被要求产生第一个想到的样例。结果扩展域中保存的启动效应，可以观察到在健忘症，并建议在正常的SS中观察到的启动功能描述的能力，是独立的大脑系统受损的健忘症。(45参考）（PsycInfo数据库记录（c）2022阿帕，保留所有权利）
Amnesia is considered to reflect the effects of damage to a specific brain system required for elaboration, consolidation, and conscious recollection. The study of amnesia is therefore considered by the authors to be a useful approach for establishing dissociations of function and for understanding the normal organization of memory functions. Two studies of priming—involving 8 39–74 yr old amnesic patients with alcoholic Korsakoff syndrome, 3 45–56 yr old males with amnesia resulting from hypotensive episodes, 18 37–63 yr old alcoholic males (controls), and 16 25–61 yr old normal controls—were conducted. In Exp I, as measured by a word-completion test, all groups exhibited significant priming effects that were greater within a modality than across modalities. Amnesic Ss exhibited normal priming effects both within and across modalities, despite severe impairment in recall. In Exp II, all groups exhibited significant and equivalent priming of category exemplars when category labels were presented, and Ss were asked to produce the 1st exemplars that came to mind. Results extend the domain in which preserved priming effects can be observed in amnesia and suggest that features of priming observed in normal Ss describe a capacity that is independent of the brain system damaged in amnesia.(45 ref)(PsycInfo Database Record (c) 2022 APA, all rights reserved)