Linking pattern recognition and salicylic acid responses in Arabidopsis through ACCELERATED CELL DEATH6 and receptors.
Linking pattern recognition and salicylic acid responses in Arabidopsis through ACCELERATED CELL DEATH6 and receptors.
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通过加速细胞死亡 6 和受体将拟南芥中的模式识别和水杨酸反应联系起来。
DOI：
10.1080/15592324.2015.1010912
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发表时间：
2015
影响因子：
2.9
通讯作者：
Greenberg,JeanT
中科院分区：
文献类型：
--
作者：
Tateda,Chika;Zhang,Zhongqin;Greenberg,JeanT
拟南芥膜蛋白加速细胞死亡6（ACD 6）和防御信号水杨酸（SA）是正反馈环的一部分，调节至少2种病原体相关分子模式（PAMP）受体的水平，包括鞭毛蛋白传感2（FLS 2）和几丁质激发受体（LYSM结构域受体样激酶1，CERK 1）。这些受体的ACD 6和SA介导的调节导致对FLS 2和CERK 1配体（例如分别为flg 22和几丁质）的应答增强。即使在不存在PAMP的情况下，ACD 6、FLS 2和CERK 1对于响应于SA激动剂的胼胝质诱导也是重要的。在这里，我们报告了另一种受体，EF-Tu受体（EFR）也是ACD 6/SA信号网络的一部分，类似于FLS 2和CERK 1。
TheArabidopsismembrane protein ACCELERATED CELL DEATH 6 (ACD6) and the defense signal salicylic acid (SA) are part of a positive feedback loop that regulates the levels of at least 2 pathogen-associated molecular patterns (PAMP) receptors, including FLAGELLIN SENSING 2 (FLS2) and CHITIN ELICITOR RECEPTOR (LYSM domain receptor-like kinase 1, CERK1). ACD6- and SA-mediated regulation of these receptors results in potentiation of responses to FLS2 and CERK1 ligands (e.g. flg22 and chitin, respectively). ACD6, FLS2 and CERK1 are also important for callose induction in response to an SA agonist even in the absence of PAMPs. Here, we report that another receptor, EF-Tu RECEPTOR (EFR) is also part of the ACD6/SA signaling network, similar to FLS2 and CERK1.