Effect of Permanent Magnet Stirring on Solidification of High-sulfur Micro-alloyed Steel under Different Magnetic Flux Densities

Effect of Permanent Magnet Stirring on Solidification of High-sulfur Micro-alloyed Steel under Different Magnetic Flux Densities
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不同磁通密度下永磁搅拌对高硫微合金钢凝固的影响

DOI:
10.1002/srin.202200635
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发表时间:
2022
影响因子:
2.2
通讯作者:
Jie Zeng
Jie Zeng
中科院分区:
材料科学3区
文献类型:
--
作者:
Jianfei Peng;Wanlin Wang;Wenhao Zhou;Jie Zeng

文献摘要

相似文献

通过分析MnS析出相、显微组织和拉伸性能,研究了不同中心磁场强度(0、850、1450和1800 Gs)的永磁搅拌(PMS)对高硫微合金化49 MnVS 3钢凝固过程的影响。结果表明,随着磁场强度的增加,熔体中的湍流增强,MnS析出相不受晶界限制,在钢中随机均匀分布。当PMS的磁通密度从0增加到1800 Gs时,MnS的平均长度从6.8 μm减小到2.9 μm,MnS的数密度从1562增加到2293 n mm−2。同时,晶内铁素体(IGF)的含量从0.4%增加到3.1%,IGF占总铁素体的比例从4.2%显著增加到30.1%,这是由于PMS后更细的MnS-(Nb,V,Ti)(C,N)复合夹杂物的析出导致IGF的形成。此外,在具有增强的磁通密度的PMS下,钢的抗拉强度和韧性均得到改善,这归因于小尺寸MnS析出物的增加并促进IGF的形成。
The effects of permanent magnet stirring (PMS) with different center magnetic flux densities (0, 850, 1450, and 1800 Gs) on the solidification process of high‐sulfur micro‐alloyed 49MnVS3 steel are investigated by analyzing the MnS precipitation, microstructure, and tensile properties. The results indicate that with an increase in the magnetic flux density, an enhanced turbulent flow is introduced to the melt, and the MnS precipitates are not limited by grain boundary and distributed randomly and uniformly in the steel. When the magnetic flux density of PMS increases from 0 to 1800 Gs, the mean length of MnS decreases from 6.8 to 2.9 μm, and the number density of MnS increases from 1562 to 2293 n mm−2. Meanwhile, the content of the intragranular ferrite (IGF) increases from 0.4% to 3.1%, and the ratio of IGF in the total ferrite increases significantly from 4.2% to 30.1%, due to the growing precipitation of finer MnS‐ (Nb, V, Ti) (C, N) complex inclusions after PMS that induces the formation of IGF. In addition, both the tensile strength and toughness of the steel are improved under PMS with enhanced magnetic flux densities, which is attributed to the increased small‐sized MnS precipitates and promotes the formation of IGF.