Castration and erection. An animal study.
Castration and erection. An animal study.
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阉割和勃起。
DOI：
10.1159/000473410
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发表时间：
1988
期刊：
影响因子：
23.4
通讯作者：
E. Tanagho
中科院分区：
文献类型：
--
作者：
S. C. Müller;J. Hsieh;T. Lue;E. Tanagho
与非去势动物（n = 24）相比，去势犬（n = 3）需要更高的能量阈值水平才能通过电刺激海绵体神经诱导勃起。此外，以最大海绵体内压（pCC）与收缩压峰值（BP）衡量的勃起质量，去势犬（pCC =平均BP的57%）比非去势犬（pCC =平均BP的80%）弱。去势犬阴茎海绵体的静脉流出量高也可以解释勃起持续时间短的原因。该实验模型排除了主观因素的干扰，如性刺激和性欲对勃起的影响，并且似乎雄激素缺乏对勃起组织的神经生理学有直接影响，导致消肿因子的张力更高，这可以通过窦平滑肌的不完全松弛来解释。
Castrated dogs (n = 3) need a much higher threshold level of energy to induce erection by electrical stimulation of the cavernous nerve than noncastrated animals (n = 24). In addition the resulting quality of erection, measured as maximal intracavernous pressure (pCC) versus peak systolic blood pressure (BP), was weaker in castrated dogs (pCC = 57% of BP on average) than in noncastrated dogs (pCC = 80% of BP on average). A high venous outflow from the corpora cavernosa in castrated dogs can also explain the shorter duration of erection. This experimental model excludes the interference of subjective factors, such as erotic stimuli and libido on erection, and it seems that androgen deficiency has a direct effect on the neurophysiology of the erectile tissues resulting in a higher tonus of the detumescence factors, which can be explained by an incomplete relaxation of the sinusoidal smooth muscle.