Comment on “CD226 Is Specifically Expressed on the Surface of Th1 Cells and Regulates Their Expansion and Effector Functions”
Comment on “CD226 Is Specifically Expressed on the Surface of Th1 Cells and Regulates Their Expansion and Effector Functions”
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DOI：
10.4049/jimmunol.176.7.3855
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发表时间：
2006-04
期刊：
影响因子：
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通讯作者：
K. Shibuya;K. Shibata;S. Tahara-Hanaoka;Akira Shibuya
中科院分区：
文献类型：
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作者：
K. Shibuya;K. Shibata;S. Tahara-Hanaoka;Akira Shibuya
久保和石泽博士对我们的研究提出了两个问题，这些研究证明了TLR4−/−高氧性肺损伤的易感性增加([1][1])。提出的第一个问题是，具有TLR4表达缺陷小鼠的C3H/HeJ小鼠对高氧具有抵抗力([2][2])，这是
Doctors Kubo and Ishizawa raise two issues regarding our studies demonstrating the increased susceptibility of TLR4 −/− hyperoxic lung injury ([1][1]). The first issue raised is the fact that C3H/HeJ mice, which have mice to defective TLR4 expression, are resistant to hyperoxia ([2][2]), which