A new pathotype of Pea seedborne mosaic virus explained by properties of the p3-6k1-and viral genome-linked protein (VPg)-coding regions
A new pathotype of Pea seedborne mosaic virus explained by properties of the p3-6k1-and viral genome-linked protein (VPg)-coding regions
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DOI：
10.1094/mpmi.2002.15.2.169
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发表时间：
2002-02-01
影响因子：
3.5
通讯作者：
Johansen, IE
中科院分区：
文献类型：
--
作者：
Hjulsager, CK;Lund, OS;Johansen, IE
豌豆种传花叶病毒的第四个致病型，马铃薯Y病毒属的成员，通过对一组豌豆株系的感染谱的分析来鉴定。命名为P-3的新致病型能够克服由sbm-1抗性基因指定的抗性，但不能克服由sbm-2抗性基因指定的抗性。这种感染特征将P-3与先前描述的病理型P-1、P-2和P-4区分开来。嵌合病毒的分析表明，P3- 6 k1和病毒基因组连接蛋白（VPg）编码区的特性占新的致病型的感染概况。
A fourth pathotype of Pea seedborne mosaic virus, a member of the genus Potyvirus, was identified by analysis of the infection profile on a panel of Pisum sativum lines. The new pathotype, designated P-3, was able to overcome resistance specified by the sbm-1 resistance gene but could not overcome resistance specified by the sbm-2 resistance gene. This infection profile distinguished P-3 from previously described pathotypes, P-1, P-2, and P-4. Analysis of chimeric viruses demonstrated that properties of the P3-6k1- and viral genome-linked protein (VPg)-coding regions accounted for the infection profile of the new pathotype.