Autoimmunization and Lymphomagenesis in Parent‐F1 Combinations Differing at the Major Histocompatibility Complex: Model for Spontaneous Disease Caused by Altered Self‐Antigens?
Autoimmunization and Lymphomagenesis in Parent‐F1 Combinations Differing at the Major Histocompatibility Complex: Model for Spontaneous Disease Caused by Altered Self‐Antigens?
复制标题
亲本 F1 组合的自身免疫和淋巴瘤发生在主要组织相容性复合物上有所不同：自身抗原改变引起的自发性疾病模型？
DOI：
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发表时间：
1976
影响因子：
4
通讯作者：
Wolfdietrich Wilke
中科院分区：
文献类型：
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作者：
E. Gleichmann;H. Gleichmann;Wolfdietrich Wilke
二.慢性GVHR中的淋巴瘤发生临床病程和病理学B。诱导恶性淋巴瘤需要F1宿主中的I区差异。GVHR诱导的恶性淋巴瘤的可移植性D.宿主和供者型淋巴瘤E.免疫抑制和GVHR诱导的恶性淋巴瘤之间缺乏相关性F。病毒和GVHR诱导的恶性淋巴瘤G.供体淋巴细胞在F1宿主体内的存留及其与淋巴瘤发生H的关系。GVHR诱发恶性淋巴瘤发病机制探讨1. 2.第二次世界大战宿主型淋巴瘤
II. Lymphomagenesis in the Chronic GVHR A. Clinical Course and Pathology B. Requirement of an I-Region DiHerence in the Fl Host for Induction of Malignant Lymphomas C. Transplantahility of GVHR-induced Malignant Lymphomas D. Determination of Hostand Donor-Type Lymphomas E. Lack of Correlation between Immunosuppression and GVHR-induced Malignant Lymphomas F. Viruses and GVHR-induced Malignant Lymphomas G. Persistence of Donor Lymphocytes in the Fl Host and Their Relationship to Lymphomagenesis H. Discussion of Pathogenesis of GVHRinduced Malignant Lymphomas 1. Donor-Type Lymphomas 2. Host-Type Lymphomas