Peripheral nerve compression by brachial artery-basilic vein vascular access in long-term hemodialysis.
Peripheral nerve compression by brachial artery-basilic vein vascular access in long-term hemodialysis.
复制标题
长期血液透析中肱动脉-贵要静脉血管通路压迫周围神经。
DOI：
10.1136/bmj.3.5776.706-a
复制
发表时间：
1984
影响因子：
4.3
通讯作者：
W. H. Baugher
中科院分区：
文献类型：
--
作者：
L. Reinstein;W. P. Reed;J. Sadler;W. H. Baugher
本报告首次描述了接受长期血液透析的尿毒症患者肱动脉贵要静脉血管通路的并发症，即直接周围神经压迫。介绍了三名患者。1例患者贵要静脉血肿扩张压迫桡神经。在第二名患者中，肱动脉附近的移植物相关脓肿压迫正中神经。第三例患者贵要静脉瘤压迫正中神经。在两个案例中，电诊断研究有助于确定周围神经功能障碍的位置和严重程度，并排除更常见的远端神经功能障碍，这是有时在这些患者中看到的透析引起的肢体循环改变的结果。及时手术减压导致所有三名患者的神经功能显着恢复。结论：直接周围神经压迫是尿毒症患者长期血液透析肱动脉贵要静脉血管通路的罕见并发症。
This report presents the first description of direct peripheral nerve compression as a complication of brachial artery-basilic vein vascular access in patients with uremia who are receiving long-term hemodialysis. Three patients are presented. An expanding basilic vein hematoma compressed the radial nerve in one patient. A graft associated abscess adjacent to the brachial artery compressed the median nerve in a second patient. A basilic vein aneurysm compressed the median nerve in a third patient. In two cases, electrodiagnostic studies were helpful in identifying the location and severity of the peripheral nerve dysfunction, and in excluding more common distal nerve dysfunction, which is sometimes seen in these patients as a result of circulatory alterations in the extremity caused by dialysis. Prompt surgical decompression led to significant recovery of nerve function in all three patients. It is concluded that direct peripheral nerve compression is a rare complication of brachial artery-basilic vein vascular access in patients with uremia who are receiving long-term hemodialysis.