Recombinational joints in a simian virus 40 variant generated in a persistent infection.
Recombinational joints in a simian virus 40 variant generated in a persistent infection.
复制标题
持续感染中产生的猿猴病毒 40 变种的重组关节。
DOI：
10.1099/0022-1317-63-2-517
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发表时间：
1982
期刊：
影响因子：
--
通讯作者：
Piatak,M
中科院分区：
文献类型：
--
作者：
Norkin,LC;Piatak,M
SP1, a viable simian virus 40 (SV40) variant isolated from a persistent infection of rhesus monkey kidney cells, contains sequence rearrangements in the untranslated region of the SV40 genome which are transcribed into late mRNA leader sequences and in the region which encodes the large T antigen. Nucleotide sequences about the recombinational junctions in SP1 were determined. The sequence data show that in most instances there was not extensive homology between recombining sequences. The recombinant sequences are discussed with respect to the mechanisms by which they might have been generated.
