Oxidized ATM promotes the abnormal proliferation of breast CAF through keeping intracellular redox homeostasis and activating the PI3K-AKT, ERK-MAPK, and Wnt signaling pathways.
Oxidized ATM promotes the abnormal proliferation of breast CAF through keeping intracellular redox homeostasis and activating the PI3K-AKT, ERK-MAPK, and Wnt signaling pathways.
复制标题
氧化ATM通过维持细胞内氧化还原稳态并激活PI3K-AKT、ERK-MAPK和Wnt信号通路促进乳腺CAF的异常增殖。
作者：
Shifu Tang;Yixuan Hou;Hailong Zhang;Gang Tu;Li Yang;Yifan Sun;Lei Lang;Xi Tang;Yan-e Du;Mingli Zhou;Tenghua Yu;Liyun Xu;Siyang Wen;Chunming Liu;Manran Liu
