Acute small-bowel obstruction following mesenteric perforation by CAPD catheter.
Acute small-bowel obstruction following mesenteric perforation by CAPD catheter.
复制标题
CAPD 导管肠系膜穿孔后急性小肠梗阻。
DOI：
10.1093/ndt/12.3.599
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发表时间：
1997
期刊：
影响因子：
--
通讯作者：
A. Davison
中科院分区：
文献类型：
--
作者：
N. S. Kanagasundaram;S. Fletcher;S. Gupta;A. Davison
患者为一名70岁白人男性，患有继发于高血压肾硬化的终末期肾衰竭，于1994年4月通过直视下经横向切口插入Oreopoulos导管进入骨盆开始CAPD。同时行左腹股沟疝修补术。CAPD并发一次葡萄球菌性腹膜炎，完全用腹膜内万古霉素治疗（1995年1月），和两次便秘发作（1994年7月/12月），需要住院治疗。两次发作均以腹痛和呕吐为特征，但腹膜白色细胞计数和培养正常，腹部平片证实粪便负荷，无肠梗阻证据。第二次发作提示腹部计算机断层扫描和钡灌肠，其中唯一的异常检测是乙状结肠憩室。CAPD在接下来的12年中进展顺利
The patient, a 70-year-old caucasian male with endstage renal failure secondary to hypertensive nephrosclerosis, commenced CAPD in April 1994 via an Oreopoulos catheter inserted through a transverse incision under direct vision into the pelvis. Concomitant left inguinal herniorrhaphy was performed. CAPD was complicated by one bout of staphylococcal peritonitis, fully treated with intraperitoneal vancomycin (January 1995), and two episodes (July/December 1994) of constipation requiring hospital admission. Each of the two episodes was marked by abdominal pain and vomiting but associated with normal peritoneal white cell counts and cultures, and plain abdominal radiographs confirming faecal loading with no evidence of intestinal obstruction. The second episode prompted an abdominal computerized tomographic scan and barium enema from which the only abnormality detected were sigmoid diverticula. CAPD had progressed uneventfully for the next 12