Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in in vitro model of preeclamptic vascular endothelium : apossible therapeutic role of nicotinic acetylcholine receptor(nAChR)agoonists for preeclampsia.
Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in in vitro model of preeclamptic vascular endothelium : apossible therapeutic role of nicotinic acetylcholine receptor(nAChR)agoonists for preeclampsia.
复制标题
尼古丁恢复子痫前期血管内皮体外模型中过量 sFlt1 和 sEng 引起的内皮功能障碍：烟碱乙酰胆碱受体 (nAChR) 激动剂对先兆子痫的可能治疗作用。
作者：
Mimura K;Tomimatsu T
