Staphylococcus aureus cap5O and cap5P genes functionally complement mutations affecting enterobacterial common-antigen biosynthesis in Escherichia coli.
Staphylococcus aureus cap5O and cap5P genes functionally complement mutations affecting enterobacterial common-antigen biosynthesis in Escherichia coli.
复制标题
金黄色葡萄球菌 cap5O 和 cap5P 基因功能性地补充影响大肠杆菌中肠杆菌共同抗原生物合成的突变。
DOI：
10.1128/jb.180.2.403-406.1998
复制
发表时间：
1998
影响因子：
3.2
通讯作者：
Lee,JC
中科院分区：
文献类型：
--
作者：
Kiser,KB;Lee,JC
TheStaphylococcus aureus cap5Pandcap5Ogenes of the type 5 capsule biosynthetic locus restore enterobacterial common-antigen expression toEscherichia colimutants defective inrffEandrffDgene expression, respectively. Cap5P and Cap5O likely function as UDP-GlcNAc 2-epimerase and UDP-ManNAc dehydrogenase enzymes, respectively, in the synthesis of the capsule precursor UDP-ManNAcA.
