Maternal nutrition, placental growth and fetal programming
Maternal nutrition, placental growth and fetal programming
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DOI：
10.1079/pns19980016
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发表时间：
1998-02-01
影响因子：
7
通讯作者：
Robinson, S
中科院分区：
文献类型：
--
作者：
Godfrey, K;Robinson, S
出生时身材偏小或不成比例，或者胎盘生长发生改变的婴儿在成年后患冠心病、高血压和非胰岛素依赖型糖尿病的几率会增加。这些关联被认为反映了胎儿的“编程”。尽管人们对母体对人类胎儿的影响知之甚少，但初步研究指出，母亲通过胎盘供应线满足胎儿营养需求的能力非常重要。
Babies who are small or disproportionate at birth, or who have altered placental growth have increased rates of CHD, hypertension and non-insulin-dependent diabetes in adult life. These associations are thought to reflect fetal ‘programming’. Although little is known about the maternal influences which programme the human fetus, initial studies point to the importance of a mother’s capacity to satisfy nutrient requirements of her fetus through the placental supply line.