Role of Host-Specific Toxins in the Pathogenesis of Alternaria alternata
Role of Host-Specific Toxins in the Pathogenesis of Alternaria alternata
复制标题
宿主特异性毒素在链格孢发病机制中的作用
DOI：
10.1007/978-1-4612-3084-7_12
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发表时间：
1991
期刊：
影响因子：
--
通讯作者：
S. Nishimura
中科院分区：
文献类型：
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作者：
H. Otani;K. Kohmoto;M. Kodama;S. Nishimura
寄主识别及其在寄主-寄生物相互作用中的特异性是植物生理病理学研究领域中最具吸引力的课题。最近，已经提出了几个模型，负责确定疾病特异性的机制。这些模型之一来自真菌病原体产生的宿主特异性或宿主选择性毒素（HST）的研究。确定涉及HST的疾病中的特异性的机制由三个基本过程组成（Nishimura等人，1983; Kohmoto等人，1987，1989）：1）真菌寄生物的孢子在萌发时释放HST（宿主识别因子），2）释放的信号因子选择性地结合宿主细胞中的受体位点，和3）宿主细胞对可能的菌丝入侵的可接近状态或易感性通过信号转导来处理。
Host recognition and its specificity in host-parasite interactions are the most attractive subjects in the field of physiological plant pathology. Recently, several models have been proposed as to the mechanisms responsible for determining disease specificity. One of these models comes from studies of host-specific or host-selective toxins (HSTs) produced by fungal pathogens. A mechanism that determines specificity in the diseases involving HST is comprised of three basic processes (Nishimura et al., 1983; Kohmoto et al., 1987, 1989): 1) Spores of a fungal parasite release HST, a host recognition factor, on germination, 2) the released signal factor selectively binds to receptor sites in the host cells, and 3) the accessible state or susceptibility of host cells to possible hyphal invasion is disposed by the signal transduction.