Dissociation of FKBP12.6 from ryanodine receptor type 2 is regulated by cyclic ADP-ribose but not beta-adrenergic stimulation in mouse cardiomyocytes.
Dissociation of FKBP12.6 from ryanodine receptor type 2 is regulated by cyclic ADP-ribose but not beta-adrenergic stimulation in mouse cardiomyocytes.
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FKBP12.6 与 2 型兰尼碱受体的解离受小鼠心肌细胞中环 ADP 核糖的调节,但不受 β 肾上腺素能刺激的调节。
DOI:
10.1093/cvr/cvp212
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发表时间:
2009-11
影响因子:
10.8
通讯作者:
中科院分区:
文献类型:
--
作者:
AIMS Beta-adrenergic augmentation of Ca(2+) sparks and cardiac contractility has been functionally linked to phosphorylation-dependent dissociation of FK506 binding protein 12.6 (FKBP12.6) regulatory proteins from ryanodine receptors subtype 2 (RYR2). We
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