Activation of agonist and antagonist muscles at different joint angles during maximal isometric efforts
Activation of agonist and antagonist muscles at different joint angles during maximal isometric efforts
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DOI：
10.1007/s00421-003-1025-x
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发表时间：
2004-03-01
影响因子：
3
通讯作者：
Fukunaga, T
中科院分区：
文献类型：
--
作者：
Kubo, K;Tsunoda, N;Fukunaga, T
本研究的目的是调查不同角度的膝关节的激动剂（股四头肌）和拮抗剂（股二头肌）的激活水平的影响，从肌电活动和激活水平（抽搐插值）。对23名健康受试者在膝关节屈曲40 °至110 °之间每隔10 °进行等长扭矩测量。施加最大随意收缩的叠加抽搐，并估计随意激活。为了量化拮抗肌活动，我们将其在每个关节角度的积分EMG（iEMG）值相对于其在最大努力时作为激动剂时在相同角度的iEMG值进行标准化。屈膝位（80- 110 °）的激活水平高于伸膝位（40- 70 °）。90 °、100 °、110 °的共激活水平显著高于其他膝关节角度。这些结果表明，激动剂（股四头肌）肌肉的激活水平和拮抗剂（股二头肌）肌肉的共激活水平在较长的肌肉中比在较短的肌肉中更高。还得出结论，膝关节损伤的风险可以通过应用这些相互关系之间的激动剂和拮抗剂肌肉的激活水平降低。
The purpose of this study was to investigate the influence of different angles of the knee joint on the activation level of an agonist (quadriceps femoris muscle) and antagonist (biceps femoris muscle) from electromyographic activities and activation levels (twitch interpolation). Isometric torque measurements were performed on 23 healthy subjects at 10degrees intervals between 40degrees and 110degrees of knee joint flexion. Superimposed twitches at maximal voluntary contraction were applied and the voluntary activation estimated. To quantify the antagonist muscle activity, we normalized its integrated EMG (iEMG) value at each joint angle with respect to its iEMG value at the same angle when acting as an agonist at maximal effort. The activation levels at the knee-flexed position (80-110degrees) were higher than that at the knee-extended position (40-70degrees). The co-activation levels at 90, 100, and 110degrees were significantly higher than that the other knee angle. These results suggest that the activation level of an agonist (quadriceps femoris) muscle and the co-activation level of an antagonist (biceps femoris) muscle were higher in longer muscles than in shorter muscles. It was also concluded that the risk of knee injuries could be reduced by applying these mutual relationships between activation levels of agonist and antagonist muscles.