Induced systemic resistance to blue mold: early induction and accumulation of β-1,3-glucanases, chitinases, and other pathogenesis-related proteins (b-proteins) in immunized tobacco
Induced systemic resistance to blue mold: early induction and accumulation of β-1,3-glucanases, chitinases, and other pathogenesis-related proteins (b-proteins) in immunized tobacco
复制标题
诱导对蓝霉病的全身抗性：免疫烟草中 β-1,3-葡聚糖酶、几丁质酶和其他发病机制相关蛋白（b 蛋白）的早期诱导和积累
DOI：
10.1094/phyto-79-979
复制
发表时间：
1989
期刊：
影响因子：
3.2
通讯作者：
J. Kuc
中科院分区：
文献类型：
--
作者：
S. Tuzun;M. N. Rao;U. Vogeli;C. Schardl;J. Kuc
攻击后仅 6 天，对照组中就检测到 b 蛋白增加。总是检测到几丁质酶的基础水平，但在免疫植物中几丁质酶的增加高于该水平遵循与 β-1,3-葡聚糖酶和其他 b 蛋白相似的特征。 β-1,3-葡聚糖酶、几丁质酶和其他 b 蛋白很可能在烟草中受到协调调节。这些蛋白质的增加与注射烟粉虱的植物的免疫开始一致，并且在攻击后烟粉虱发育受到限制的时期内维持水平
Increases in b-proteins were detected in controls only 6 days after challenge. A basal level of chitinases was always detected, but increases in chitinases above this level in immunized plants followed a profile similar to that of the β-1,3-glucanases and other b-proteins. It is likely that β-1,3-glucanases, chitinases, and other b-proteins are coordinately regulated in tobacco. The increases in these proteins coincided with the onset of immunization in plants injected with P. tabacina, and the levels were maintained during the period after challenge, when the development of P. tabacina was restricted