Molecular Pathogenesis of Genetic and Inherited Diseases Disrupted Membrane Homeostasis and Accumulation of Ubiquitinated Proteins in a Mouse Model of Infantile Neuroaxonal Dystrophy Caused by PLA 2 G 6 Mutations

Molecular Pathogenesis of Genetic and Inherited Diseases Disrupted Membrane Homeostasis and Accumulation of Ubiquitinated Proteins in a Mouse Model of Infantile Neuroaxonal Dystrophy Caused by PLA 2 G 6 Mutations
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发表时间:
2010
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通讯作者:
I. Malik;J. Turk;D. Mancuso;L. Montier;M. Wohltmann;D. Wozniak;R. Schmidt;R. Gross;P. Kotzbauer
I. Malik;J. Turk;D. Mancuso;L. Montier;M. Wohltmann;D. Wozniak;R. Schmidt;R. Gross;P. Kotzbauer
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其他
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作者:
I. Malik;J. Turk;D. Mancuso;L. Montier;M. Wohltmann;D. Wozniak;R. Schmidt;R. Gross;P. Kotzbauer

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来自圣路易斯华盛顿大学医学院神经病学系、* 内分泌、代谢和脂质研究以及医学系生物有机化学和分子药理学系、精神病学系、病理学和免疫学系、神经疾病霍普中心以及分子生物学和药理学系;和密苏里州圣路易斯市的华盛顿大学化学系
From the Department of Neurology,* the Divisions of Endocrinology, Metabolism, and Lipid Research and Bioorganic Chemistry and Molecular Pharmacology in the Department of Medicine, the Departments of Psychiatry and Pathology and Immunology, Hope Center for Neurological Disorders, and the Department of Molecular Biology and Pharmacology,** Washington University School of Medicine, St. Louis; and the Department of Chemistry, Washington University, St. Louis, Missouri