Augmented hyperventilation via normoxic helium breathing does not prevent exercise-induced hypoxemia.
Augmented hyperventilation via normoxic helium breathing does not prevent exercise-induced hypoxemia.
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通过常氧氦气呼吸增强过度通气并不能预防运动引起的低氧血症。
DOI：
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发表时间：
1996
期刊：
影响因子：
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通讯作者：
R. Maly
中科院分区：
文献类型：
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作者：
M. Buono;R. Maly
本研究的目的是确定通过常氧氦呼吸产生的增强过度通气是否会减少运动引起的低氧血症（EIH）。七名受过严格训练的耐力运动员，平均最大摄氧量为65 ml·kg ~（-1）·min ~（-1），进行了两次自行车测力计测试，以达到意志衰竭。在其中一项测试中，受试者呼吸环境空气，而在另一项测试中，他们呼吸含氧量正常的氦气（21% O2，79% He）。平均最大呼气通气量显著（p <0.05）从环境试验期间的139 L.min-1增加到呼吸常氧氦时的168 L.min-1。然而，在最大运动量下获得的平均动脉血氧饱和度在两项试验中没有显著差异（环境= 90%，氦气= 89%）。这些结果表明，显著增加运动过度通气21%基本上对耐力运动员的EIH没有影响。因此，这些数据不支持这样的假设，即过度通气不足是高度训练的个体在分级运动至力竭期间动脉氧饱和度下降的重要机制。
The purpose of this study was to determine if augmented hyperventilation produced via normoxic helium breathing would reduce exercise-induced hypoxemia (EIH). Seven highly trained endurance athletes with a mean maximum oxygen uptake of 65 ml.kg-1.min-1, performed two cycle ergometer tests to volitional exhaustion. During one of the tests the subjects breathed ambient air, while during the other they breathed normoxic helium (21% O2, 79% He). Mean maximum expired ventilation significantly (p < .05) increased from 139 L.min-1 during the ambient trial to 168 L.min-1 while breathing normoxic helium. Mean arterial oxygen saturation obtained at maximum exercise, however, was not significantly different for the two trials (ambient = 90%, helium = 89%). These results suggest that significantly augmenting exercise hyperventilation by 21% essentially had no effect on EIH in endurance athletes. Thus, the data do not support the hypothesis that inadequate hyperventilation is an important mechanism for arterial oxygen desaturation during graded exercise to exhaustion in highly trained individuals.