Assessment of the role of the cochlear latency effect in lateralization of click sounds in humans
Assessment of the role of the cochlear latency effect in lateralization of click sounds in humans
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DOI：
10.1111/j.1469-8986.2009.00828.x
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发表时间：
2009-07-01
期刊：
影响因子：
3.7
通讯作者：
Ungan, Pekcan
中科院分区：
文献类型：
--
作者：
Ozmen, Buelent;Ungan, Pekcan
两耳间的时间和强度差异（ITD和IID）是声音偏侧化的两个线索。“仅时间”假说声称，IID首先转换为耳间传入延迟（Delta t），然后由中央ITD机制处理，从而无需单独的IID处理器。我们通过评估耳蜗潜伏期效应对心理物理ITD/IID交易比率的贡献来测试这一假设。听觉脑干反应（ABR）被用来测量在交易实验中使用的20/秒的双耳分听点击训练所产生的耳间传入延迟（Delta ts）。除了在低响度水平的小IID，由IID产生的生理Δ t延迟是显着小于ITD精神病交易相同的IID。我们的结论是，耳蜗潜伏期的影响不能单独解释的心理物理ITD/IID交易比率和一个单独的IID机制必须参与。
Interaural time and intensity disparities (ITD and IID) are the two cues to sound lateralization. "Time-only'' hypothesis claims that an IID is first converted to an interaural afferent delay (Delta t), and is then processed by the central ITD mechanism, rendering a separate IID processor unnecessary. We tested this hypothesis by assessing the contribution of the cochlear latency effect to the psychophysical ITD/IID trading ratio. Auditory brainstem responses (ABRs) were used to measure the interaural afferent delays (Delta ts) that developed with a 20/sec dichotic click train used in the trading experiment. Except for small IIDs at low loudness levels, the physiological Delta t delay produced by an IID was significantly smaller than the ITD psychophysically traded for the same IID. We concluded that the cochlear latency effect alone cannot explain the psychophysical ITD/IID trading ratios and a separate IID mechanism must be involved.