Paroxysmal supraventricular tachycardia with Wenckebach block: evidence for reentry within the upper portion of the atrioventricular node.
Paroxysmal supraventricular tachycardia with Wenckebach block: evidence for reentry within the upper portion of the atrioventricular node.
复制标题
阵发性室上性心动过速伴温克巴赫传导阻滞：房室结上部折返的证据。
DOI：
10.1016/s0735-1097(84)80295-8
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发表时间：
1984
影响因子：
24
通讯作者：
Belardinelli,L
中科院分区：
文献类型：
--
作者：
DiMarco,JP;Sellers,TD;Belardinelli,L
阵发性室上性心动过速伴温克巴赫传导阻滞是一种罕见的心电图表现。对表现出这种现象的患者进行的电生理研究表明，心动过速不是由窦房结或心房折返或异常自动性引起的。施用阿托品后心动过速周期长度缩短，并响应普萘洛尔而增加。给予腺苷（75 至 112.5 µ/kg）或维拉帕米（10 毫克）可终止个别心动过速发作。在维拉帕米后，当引入心房额外刺激时，可以观察到心房活动与心室活动的分离，尽管也可能发生具有明显温克巴赫周期性的房室（AV）结块传导阻滞。这些发现表明，阵发性室上性心动过速可能是由仅位于房室结上部的折返引起的。
Paroxysmal supraventricular tachycardia with wenckebach block is an uncommon electrocardiographic finding. Electrophysiologic studies performed in a patient who exhibited this phenomenon indicated that the tachycardia was not caused by either sinus node or intraatrial reentry or abnormal automaticity. The tachycardia cycle length decreased after atropine administration and increased in response to propranolol. Administration of either adenosine (75 to 112.5 µ/kg) or verapamil (10mg) terminated individual episodes of tachycardia. After verapamil and when atrial extrastimuli were introduced, dissociation of atrial activity from His-ventricular activity could be observed even though atrioventricular (AV) nodal block with a clear Wenckebach periodicity could also occur. These findings suggest that paroxysmal Supraventricular tachycardia may be produced by reentry located solely within the upper portion of the AV node.
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影响因子：
39.2
作者：
DiMarco,JP;Garan,H;Harthorne,JW;Ruskin,JN
通讯作者：
Ruskin,JN
影响因子：
--
作者：
P. Gupta;E. Lichstein;K. Chadda
通讯作者：
K. Chadda
房室结性心动过速期间的二度传导阻滞
影响因子：
37.8
作者：
H. Wellens;J. Wesdorp;Donald R. Doren;K. Lie
通讯作者：
K. Lie
影响因子：
37.8
作者：
DIMARCO, JP;SELLERS, TD;BELARDINELLI, L
通讯作者：
BELARDINELLI, L
影响因子：
37.8
作者：
L. Belardinelli;A. West;R. Crampton;R. Berne
通讯作者：
R. Berne