Neurogenic pulmonary edema and missile emboli.
Neurogenic pulmonary edema and missile emboli.
复制标题
DOI：
10.1097/00005373-198008000-00012
复制
发表时间：
1980-08
期刊：
影响因子：
--
通讯作者：
Reines Hd;L. Dill;S. Saad;Hungerford Gd
中科院分区：
文献类型：
--
作者：
Reines Hd;L. Dill;S. Saad;Hungerford Gd
本文报告一例弹丸栓子致颅内大血管暴发性肺水肿的病例。对神经源性肺水肿(NPE)的病因进行了讨论。病因被认为是由于中枢神经系统大量释放儿茶酚，导致肺动脉压力显著增加，并可能继发于毛细血管后括约肌收缩。应清除大脑动脉的飞弹栓子，以避免远端血栓和脑梗塞。报告的病例似乎是第一个记录在案的导弹栓子导致NPE的病例。
A case of missile emboli to a major intracranial vessel resulting in fulminant pulmonary edema is reported. A discussion of the etiology of neurogenic pulmonary edema (NPE) is presented. The cause is thought to be a marked increase in pulmonary artery pressure, due to a massive catechol release in the CNS, and possibly secondary to post-capillary sphincteric constriction. Missile emboli to the cerebral arteries should be removed in order to avoid distal embolization and infarction. The case reported appears to represent the first documented case of missile emboli causing NPE.