Causality orientations moderate the undermining effect of rewards on intrinsic motivation
Causality orientations moderate the undermining effect of rewards on intrinsic motivation
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DOI：
10.1016/j.jesp.2010.10.010
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发表时间：
2011-03-01
影响因子：
3.5
通讯作者：
Chatzisarantis, Nikos L. D.
中科院分区：
文献类型：
--
作者：
Hagger, Martin S.;Chatzisarantis, Nikos L. D.
本研究验证了自主性或控制性因果取向的个体差异会调节奖励对内在动机的破坏作用这一假设。相同数量的自主性和控制导向的参与者提供了一个有趣的难题下奖励或无奖励的条件下的解决方案。然后，参与者被要求在自由选择的时间内提供进一步的解决方案。在自由选择阶段，被试解决问题所花费的时间构成了内在动机的独立测量。结果表明，因果取向和奖励条件对内在动机有显著的交互作用。控制导向的参与者分配到奖励条件表现出显着较低水平的内在动机相比，那些分配到没有奖励的条件。与此相反，有没有显着差异的内在动机水平在整个奖励条件的忠诚度为导向的个人。研究结果表明，一个以利益为导向的因果关系取向提供了一定程度的“保护”的破坏性影响的奖励对内在动机。这与自我决定理论中环境事件和人际因素对内在动机的交互作用是一致的。(c)2010年爱思唯尔公司All rights reserved.
The present study tested the hypothesis that individual differences in autonomy or control causality orientation would moderate the undermining effect of rewards on intrinsic motivation. Equal numbers of autonomy- and control-orientated participants provided solutions to an interesting puzzle under reward or no reward conditions. Participants were then required to provide further solutions to the puzzle in a free-choice period. The time spent in solving the puzzle during the free-choice period constituted the dependent measure of intrinsic motivation. Results revealed a significant interaction effect of causality orientations and reward condition on intrinsic motivation. Control-oriented participants assigned to the reward condition exhibited significantly lower levels of intrinsic motivation compared to those assigned to the no reward condition. In contrast, there was no significant difference in intrinsic motivation levels across the reward conditions for autonomy-oriented individuals. Findings indicate that an autonomy-oriented causality orientation offered a degree of 'protection' from the undermining effect of rewards on intrinsic motivation. This is in keeping with self-determination theory in terms of the interactive effects of environmental events and interpersonal factors on intrinsic motivation. (c) 2010 Elsevier Inc. All rights reserved.