CALCIUM DEFICIENCY AND PLANT ULTRASTRUCTURE
CALCIUM DEFICIENCY AND PLANT ULTRASTRUCTURE
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DOI：
10.1080/00103627909366879
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发表时间：
1979-01-01
影响因子：
1.8
通讯作者：
HECHTBUCHHOLZ, C
中科院分区：
文献类型：
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作者：
HECHTBUCHHOLZ, C
通过在潮湿环境中培养马铃薯芽，诱导发育中的马铃薯芽缺钙。由于缺钙症状首先发生在顶芽下的芽伸长区，因此制备了该区域皮层的组织切片进行电镜观察。在缺钙组织中，观察到与严重钙自溶的单个细胞相邻的细胞中细胞质退化的开始阶段。细胞质的开始破裂由质膜的突起和开始解体、退化小泡的积累和液泡膜的开始解体指示。在具有钙缺乏自溶的细胞附近的相对健康的细胞中的大多数前质体不含淀粉，而在对照植物的相应组织中的大多数前质体中淀粉丰富。这被认为是由于缺钙芽生长迟缓的结果。
Calcium deficiency was induced in developing potato sprouts by growing them in a humid atmosphere.As calcium deficiency symptoms occurred first at the elongation zone of the sprouts under the apical bud, tissue pieces of the cortex of this zone were prepared for electronmicroscopic investigation.Beginning stages of cytoplasm degeneration in the calcium‐deficient tissue were observed in cells adjacent to individual cells with severe calcium autolysis. The beginning breakdown of the cytoplasm was indicated by protuberances and beginning disintegration of the plasmalemma, accumulation of degenerative vesicles, and the beginning disintegration of the tonoplast. Most of the proplastids in relatively healthy‐looking cells in the neighborhood of the cells with calcium deficiency autolysis did not contain starch, while starch was abundant in most of the proplastids in the corresponding tissue of the control plants. This was considered as a result of the retarded growth of the calcium‐deficient sprouts.