METABOLISM DURING NORMAL, FRAGMENTED, AND RECOVERY SLEEP
METABOLISM DURING NORMAL, FRAGMENTED, AND RECOVERY SLEEP
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DOI：
10.1152/jappl.1991.71.3.1112
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发表时间：
1991-09-01
影响因子：
3.3
通讯作者：
ARAND, DL
中科院分区：
文献类型：
--
作者：
BONNET, MH;BERRY, RB;ARAND, DL
在一组12名正常的年轻成年男性中，每3分钟的平均代谢数据(O2摄取量和二氧化碳排出量)在正常、实验分段和恢复睡眠的连续夜晚中获得。自然发生的唤醒和唤醒导致了基线之夜新陈代谢的典型增加。在接下来的晚上进行短暂频繁的实验唤醒会导致整晚新陈代谢显著增加，并显著降低通过早晨的表现、情绪和警觉性测试衡量的睡眠恢复，即使总的睡眠时间最小限度地减少了。在睡眠碎片之夜之后的未受干扰的恢复之夜，代谢变量与基线相比显著降低。这些数据不能用来推断睡眠期间新陈代谢增加会导致非恢复性睡眠，但伴随着觉醒而来的新陈代谢变化的方向和时间进程与这一假设是一致的。
Average metabolic data (O2 uptake and CO2 output) were obtained for each 3-min period during consecutive nights of normal, experimentally fragmented, and recovery sleep in a group of 12 normal young adult males. Naturally occurring arousals and awakenings resulted in a characteristic increase in metabolism on the baseline night. The placement of brief frequent experimental arousals on the following night resulted in significantly increased metabolism throughout the night and significantly decreased sleep restoration as measured by morning performance, mood, and alertness tests, even through total sleep time was minimally reduced. Metabolic variables were significantly decreased compared with baseline on the nondisturbed recovery night that followed the sleep fragmentation night. The data cannot be used to infer that increased metabolism during sleep causes nonrestorative sleep, but the direction and time course of metabolic change accompanying arousal are consistent with that hypothesis.