In vitro evidence for the ionoregulatory role of rainbow trout mucus in acid, acid/aluminium and zinc toxicity
In vitro evidence for the ionoregulatory role of rainbow trout mucus in acid, acid/aluminium and zinc toxicity
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虹鳟鱼粘液对酸、酸/铝和锌毒性的离子调节作用的体外证据
DOI：
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发表时间：
1989
期刊：
影响因子：
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通讯作者：
G. Romain
中科院分区：
文献类型：
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作者：
R. Handy;F. Eddy;G. Romain
虹鳟鱼体粘液用酸化蒸馏水在pH 7、5和3下透析经历离子耗竭，其在pH 3下最大，在pH 7和5之间最小。在pH值为5和7时，1 mg I−1铝（以A12（SO4）形式存在）会加剧粘液Na+损失。Al2（SO4）比AlCl3引起粘液中Na+更大的消耗。锌的致死水平（2 mg 1−1）不会消耗粘液中的Na−或K+，而铝的致死水平（1 mg 1−1）在pH值为7时则不同。结果进行了讨论的粘液中的重金属和酸毒性的离子调节作用。
Rainbow trout body mucus dialysed with acidified distilled water at pH 7,5 and 3 experienced ion depletion which was greatest at pH 3 and minimal between pH 7 and 5. Mucus Na+ loss is exacerbated in the presence of 1 mg I−1 aluminium as A12(SO4), at pH 5 and 7. Al2(SO4), causes greater depletion of Na+ from mucus than A1C13. A lethal level of zinc (2 mg 1−1) does not deplete mucus Na− or K+, unlike a lethal level of aluminium (1 mg 1−1) at pH 7. The results are discussed in terms of the ionoregulatory role of mucus in heavy metal and acid toxicity.