Residual dizziness after successful treatment of idiopathic benign paroxysmal positional vertigo originates from persistent utricular dysfunction
Residual dizziness after successful treatment of idiopathic benign paroxysmal positional vertigo originates from persistent utricular dysfunction
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DOI：
10.1080/00016489.2017.1347824
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发表时间：
2017-01-01
影响因子：
1.4
通讯作者：
Doi, Katsumi
中科院分区：
文献类型：
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作者：
Seo, Toru;Shiraishi, Ko;Doi, Katsumi
目的：探讨良性阵发性位置性眩晕小管复位术后残留眩晕与椭圆囊功能的关系。方法：对44例(纳入分析40例，排除4例)行小管复位术成功的患者进行眼前庭诱发肌源性电位检测。患者在治疗前和治疗一周后复查。结果：残留眩晕与性别、患侧、年龄、症状持续时间、复发或初始眼前庭诱发肌源性电位检查结果无关(p>；.05)。然而，残余头晕与第二次眼前庭诱发肌源性电位测试的结果显著相关(p=0.007)。结论：成功的泪小管复位术后的残余头晕可能是由持续性的椭圆形功能障碍引起的。
Objective: We used ocular vestibular evoked myogenic potentials to investigate the relationship between residual dizziness and utricular function following the canalith repositioning procedure for benign paroxysmal positional vertigo.Methods: Ocular vestibular evoked myogenic potentials were measured in 44 patients (40 included in analyses, four excluded) with successful results from the canalith repositioning procedure. The patients were examined before treatment and again one week after treatment. We analyzed how various general factors and ocular vestibular evoked myogenic potentials related to residual dizziness.Results: Residual dizziness was not related to gender, affected side, age, duration of symptoms, recurrence, or the results of the initial ocular vestibular evoked myogenic potential test (p >.05). However, residual dizziness was significantly associated with the results of the second ocular vestibular evoked myogenic potential test (p = .007).Conclusions: Residual dizziness after a successful canalith repositioning procedure may be caused by persistent utricular dysfunction.