Anthropogenic Secondary Organic Aerosols Contribute Substantially to Air Pollution Mortality
Anthropogenic Secondary Organic Aerosols Contribute Substantially to Air Pollution Mortality
复制标题
DOI：
10.5194/acp-2020-914
复制
发表时间：
2020-11
影响因子：
6.3
通讯作者：
B. Nault;B. Nault;D. Jo;D. Jo;B. McDonald;B. McDonald;P. Campuzano‐Jost;P. Campuzano‐Jost;D. Day;D. Day;Weiwei Hu;Weiwei Hu;Weiwei Hu;J. Schroder;J. Schroder;J. Schroder;J. Allan;D. Blake;M. Canagaratna;H. Coe;M. Coggon;M. Coggon;P. DeCarlo;G. Diskin;R. Dunmore;F. Flocke;A. Fried;J. Gilman;G. Gkatzelis;G. Gkatzelis;J. Hamilton;T. Hanisco;P. L. Hayes;D. Henze;A. Hodzic;A. Hodzic;J. Hopkins;Min Hu;L. G. Huey;B. Jobson;W. Kuster;A. Lewis;Meng Li;Meng Li;J. Liao;J. Liao;M. Nawaz;I. Pollack;J. Peischl;J. Peischl;B. Rappenglück;C. Reeves;D. Richter;J. Roberts;T. Ryerson;M. Shao;J. Sommers;J. Walega;C. Warneke;C. Warneke;P. Weibring;G. Wolfe;G. Wolfe;D. Young;D. Young;Bin Yuan;Qiang Zhang;J. Gouw;J. Gouw;J. Jimenez;J. Jimenez
中科院分区：
文献类型：
--
作者：
B. Nault;B. Nault;D. Jo;D. Jo;B. McDonald;B. McDonald;P. Campuzano‐Jost;P. Campuzano‐Jost;D. Day;D. Day;Weiwei Hu;Weiwei Hu;Weiwei Hu;J. Schroder;J. Schroder;J. Schroder;J. Allan;D. Blake;M. Canagaratna;H. Coe;M. Coggon;M. Coggon;P. DeCarlo;G. Diskin;R. Dunmore;F. Flocke;A. Fried;J. Gilman;G. Gkatzelis;G. Gkatzelis;J. Hamilton;T. Hanisco;P. L. Hayes;D. Henze;A. Hodzic;A. Hodzic;J. Hopkins;Min Hu;L. G. Huey;B. Jobson;W. Kuster;A. Lewis;Meng Li;Meng Li;J. Liao;J. Liao;M. Nawaz;I. Pollack;J. Peischl;J. Peischl;B. Rappenglück;C. Reeves;D. Richter;J. Roberts;T. Ryerson;M. Shao;J. Sommers;J. Walega;C. Warneke;C. Warneke;P. Weibring;G. Wolfe;G. Wolfe;D. Young;D. Young;Bin Yuan;Qiang Zhang;J. Gouw;J. Gouw;J. Jimenez;J. Jimenez
抽象。人为二次有机气溶胶（ASOA）是由人为排放的有机化合物形成的，在世界各地人口稠密地区的亚微米气溶胶中占很大比例，并导致空气质量差和过早死亡。然而，ASOA的前体来源知之甚少，减少人为有机排放可能产生的健康效益存在很大的不确定性。我们发现，在三大洲的11个城市地区的ASOA的生产与特定的挥发性有机化合物的人为反应性密切相关。不同城市ASOA生产的差异可以通过芳烃和中间和半挥发性有机化合物排放的差异来解释，这表明控制这些ASOA前体的重要性。通过改进ASOA的模型表示，我们将每年340，000例PM2.5过早死亡归因于ASOA，这比之前的研究高出一个数量级。最近提出的将死亡率归因于PM2.5的模型（全球暴露死亡率模型）的敏感性案例导致高达90万人死亡。这项研究的一个局限性是从有详细数据的区域外推到没有数据的其他区域。为了在这一领域取得进一步进展，需要在南美洲和中美洲、非洲、南亚和中东国家开展全面的空气质量运动。
Abstract. Anthropogenic secondary organic aerosol (ASOA), formed from anthropogenic emissions of organic compounds, constitutes a substantial fraction of the mass of submicron aerosol in populated areas around the world and contributes to poor air quality and premature mortality. However, the precursor sources of ASOA are poorly understood, and there are large uncertainties in the health benefits that might accrue from reducing anthropogenic organic emissions. We show that the production of ASOA in 11 urban areas on three continents is strongly correlated with the anthropogenic reactivity of specific volatile organic compounds. The differences in ASOA production across different cities can be explained by differences in the emissions of aromatics and intermediate- and semi-volatile organic compounds, indicating the importance of controlling these ASOA precursors. With an improved modeling representation of ASOA driven by the observations, we attribute 340,000 PM2.5 premature deaths per year to ASOA, which is over an order of magnitude higher than prior studies. A sensitivity case with a more recently proposed model for attributing mortality to PM2.5 (the Global Exposure Mortality Model) results up to 900,000 deaths. A limitation of this study is the extrapolation from regions with detailed data to others where data is not available. Comprehensive air quality campaigns in the countries in South and Central America, Africa, South Asia, and the Middle East are needed for further progress in this area.