Structural and functional modifications in glutamateric synapses following prolonged ethanol exposure
Structural and functional modifications in glutamateric synapses following prolonged ethanol exposure
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DOI：
10.1111/j.1530-0277.2006.00041.x
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发表时间：
2006-02-01
影响因子：
3.2
通讯作者：
Szumlinski, KK
中科院分区：
文献类型：
--
作者：
Chandler, LJ;Carpenter-Hyland, E;Szumlinski, KK
本文概述了2005年在美国加利福尼亚州圣巴巴拉举行的酒精中毒研究学会年会上发表的一次研讨会的会议记录。组织者和主席是L.贾德森·钱德勒。演讲内容如下：(1)慢性酒精暴露，N-甲基-D-天冬氨酸(NMDA)受体动力学，以及戒断超兴奋性，由Adam Hendricson，Regina Maldve和Richard Morrisett介绍，(2)酒精诱导的突触靶向NMDA受体的突触靶向与突触后密度-95的增强和脊柱大小有关，由Judson Chandler和Ezekiel Carpenter-Hyland介绍；(3)慢性酒精暴露后伏核内突触前和突触后的变化，由周峰、Youssef Sari和Richard Bell介绍；以及(4)累积物Hmer 2在酒精诱导的神经可塑性中的积极作用，由Karen Szumlinski介绍。
This article summarizes the proceedings of a symposium presented at the 2005 annual meeting of the Research Society on Alcoholism in Santa Barbara, California, USA. The organizer and chair was L. Judson Chandler. The presentations were (1) Chronic Ethanol Exposure, N-Methyl-D-Aspartate (NMDA) Receptor Dynamics, and Withdrawal Hyperexcitability, by Adam Hendricson, Regina Maldve, and Richard Morrisett, (2) Ethanol-Induced Synaptic Targeting of NMDA Receptors Is Associated With Enhanced Postsynaptic Density-95 Clustering and Spine Size, by Judson Chandler and Ezekiel Carpenter-Hyland; (3) Presynaptic and Postsynaptic Alterations in the Nucleus Accumbens Following Chronic Alcohol Exposure, by Feng Zhou, Youssef Sari, and Richard Bell;, and (4) An Active Role for Accumbens Homer2 Expression in Alcohol-Induced Neural Plasticity, by Karen Szumlinski.