Hypoxia- independent VEGF120 secretion is stimulated by increase of endogenous oxidative stress through PI3K-dependent pathways in artificially hypertrophied 3T3-L1 adipocytes: The regulation by distinct mechanism from MCP-1 via JNK and p38 MAPK pathways
Hypoxia- independent VEGF120 secretion is stimulated by increase of endogenous oxidative stress through PI3K-dependent pathways in artificially hypertrophied 3T3-L1 adipocytes: The regulation by distinct mechanism from MCP-1 via JNK and p38 MAPK pathways
复制标题
在人工肥大的 3T3-L1 脂肪细胞中，通过 PI3K 依赖性途径增加内源性氧化应激，刺激缺氧独立的 VEGF120 分泌：通过 JNK 和 p38 MAPK 途径通过与 MCP-1 不同的机制进行调节
DOI：
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发表时间：
2010
期刊：
影响因子：
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通讯作者：
H. Ishida
中科院分区：
文献类型：
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作者：
K. Takahashi;S. Yamaguchi;T. Shimoyama;T. Tanaka;K. Miyokawa;K. Handa;H. Katsuta;S. Nishida;K. Yoshimoto;H. Ishida
