Fetal renal hyperechogenicity in pathological pregnancies.
Fetal renal hyperechogenicity in pathological pregnancies.
复制标题
病理妊娠中胎儿肾脏高回声。
DOI：
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发表时间：
1997
影响因子：
2.4
通讯作者：
L. Kovács
中科院分区：
文献类型：
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作者：
A. Surányi;A. Pál;K. Streitman;S. Pintér;L. Kovács
在肾脏高回声与胎儿缺氧状态之间寻找一种关系。在28 ~ 36周期间对120例病理妊娠进行检查。所有这些妇女在检查期间均表现为肝酶水平中度升高，其中3人有病理性肾功能，4人表现为高尿酸血症。采用日立EUB-450型超声设备，3.5 MHz换能器检查胎儿肾脏的回声性。同时检查母亲的肾脏（肌酐、尿素- n、尿酸、甘油三酯、胆甾醇）、肝脏（SGOT、SGPT、GGT、胆红素）功能和血浆电解质（Na、K、Ca、Cl），并从脉动脐动脉采血测定相同参数。产后5天内随访新生儿身体状况，用相同的超声设备检查肾脏。新生儿病理性临床结局与胎儿高回声出现频率及高回声出现频率有显著相关性（χ 2检验，p < 0.01）。结果表明，在病理性妊娠中表现出肾脏高回声的胎儿需要特别仔细的产科控制和新生儿咨询。重要的是，高回声的情况下，接受围产期重症监护病房。胎儿肾高回声被认为与不良围产期结局的风险增加有关。
A relationship was sought between renal hyperechogenicity and the hypoxic state of fetuses. 120 pathological pregnancies were examined between the 28th and 36th week. All of these women exhibited moderately increased levels of hepatic enzymes, 3 of them had a pathological kidney function, and 4 of them displayed hyperuricemia during the examination period. The echogenicity of the fetal kidneys was examined with Hitachi EUB-450 ultrasound equipment with a 3.5 MHz transducer. The kidney (creatinine, urea-N, uric acid, triglyceride, cholsterin) and liver (SGOT, SGPT, GGT, bilirubin) functions and plasma electrolytes (Na, K, Ca, Cl) of the mothers were also examined and blood was collected from the pulsating umbilical artery for determination of the same parameters. After delivery, the physical condition of the neonates was followed and their kidneys were examined with the same ultrasound equipment within the first 5 days. There was a significant correlation between a pathological neonatal clinical outcome and the frequency of fetal and hyperechogenicity (chi-square test with Yates correction, p < 0.01). The results demonstrate that fetuses exhibiting renal hyperechogenicity in pathological pregnancies require particularly careful obstetric control and neonatological consultation. It is important that hyperechogenic cases be admitted to a perinatal intensive care unit. Fetal renal hyperechogenicity is considered to be associated with an enhanced risk of an adverse perinatal outcome.