Longitudinally extensive vasogenic edema following spinal cord infarction
Longitudinally extensive vasogenic edema following spinal cord infarction
复制标题
DOI：
10.1111/ncn3.12215
复制
发表时间：
2018-09-01
影响因子：
0.4
通讯作者：
Tsuji, Shoji
中科院分区：
文献类型：
--
作者：
Gondo, Takashi;Kurihara, Masanori;Tsuji, Shoji
虽然脊髓梗死后有短暂的病变扩大的报道，但纵向广泛的病变扩大超过几个椎节是极其罕见的。我们报告一个45岁的男性脊髓梗死，随后的纵向广泛的损害扩大的磁共振成像（MRI）后10天的梗塞。T2高强度病变的长度在初始MRI上为4个椎节段，但在随后的MRI上扩大到12个椎节段，伴有Lhermitte征，但其他方面未见神经功能恶化。急性期病变扩张超出脊髓前动脉区，弥散系数高，但颈套启动后10 d内病变有所改善，提示病变扩张是血管源性水肿所致。该病例表明，暂时性纵向病变扩张伴脊髓梗死可达12个椎节。在解释MRI表现时应谨慎，以避免过度治疗或继发性损伤。
Although transient lesion expansion has been reported after spinal cord infarction, longitudinally extensive lesion expanding more than several vertebral segments is extremely rare. We report a 45-year-old man having spinal cord infarction with subsequent longitudinally extensive lesion expansion on magnetic resonance imaging (MRI) 10 days after the infarction. The length of T2 hyperintense lesion was four vertebral segments on initial MRI but enlarged to span 12 vertebral segments on subsequent MRI, with Lhermitte's sign, but otherwise without neurological deterioration. The expanded lesion extended beyond the anterior spinal artery region with high apparent diffusion coefficient value in the acute phase but improved within 10 days after starting cervical collar, suggesting that lesion expansion was due to vasogenic edema. This case illustrates that transient longitudinal lesion expansion accompanied by spinal cord infarction can reach lengths of up to 12 vertebral segments. Caution should be exercised while interpreting MRI findings to avoid excessive treatment or secondary damage.