Salt and cadmium stress tolerance caused by overexpression of the Glycine Max Na+/H+ Antiporter (GmNHX1) gene in duckweed (Lemna turionifera 5511)
Salt and cadmium stress tolerance caused by overexpression of the Glycine Max Na+/H+ Antiporter (GmNHX1) gene in duckweed (Lemna turionifera 5511)
复制标题
浮萍 (Lemna turionifera 5511) 中 Glycine Max Na /H Antiporter (GmNHX1) 基因过度表达引起的盐和镉胁迫耐受性
DOI：
10.1016/j.aquatox.2017.08.010
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发表时间：
2017-11-01
影响因子：
4.5
通讯作者：
Sun, Jinsheng
中科院分区：
文献类型：
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作者：
Yang, Lin;Han, Yujie;Sun, Jinsheng
镉（Cd）污染因其毒性已引起越来越多的关注。已证实Na⁺/H⁺逆向转运蛋白（NHX1）编码一种在Na⁺/H⁺转运中已被充分研究的蛋白质，该蛋白可导致耐盐性。本研究表明，大豆Na⁺/H⁺逆向转运蛋白（GmNHX1）提高了芜萍5511的短期镉和盐抗性。GmNHX1的表达防止了根的脱落和细胞膜损伤，还能增强抗氧化系统，抑制活性氧（ROS）的积累，并在镉和盐胁迫下减少镉的吸收。对镉的耐受性表明NHX1参与了镉胁迫过程。
Cadmium (Cd) pollution has aroused increasing attention due to its toxicity. It has been proved that Na+/H+ Antiporter (NHX1) encodes a well-documented protein in Na+/H+ trafficking, which leads to salt tolerance. This study showed that Glycine max Na+/H+ Antiporter (GmNHX1) improved short-term cadmium and salt resistance in Lemna turionifera 5511. Expression of GmNHX1 prevented root from abscission and cell membrane damage, which also can enhance antioxidant system, inhibited of reactive oxygen species (ROS) accumulation and cause a less absorption of Cd under cadmium and salt stress. The cadmium tolerance suggested that NHX1 was involved under the cadmium stress.