Optimising epilepsy surgery.
Optimising epilepsy surgery.
复制标题
优化癫痫手术。
DOI:
10.1016/s1474-4422(23)00082-0
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发表时间:
2023
期刊:
影响因子:
--
通讯作者:
Duncan JS
中科院分区:
文献类型:
--
作者:
Duncan JS
7 Joly-Amado A, Hunter J, Quadri Z, et al. CCL2 Overexpression in the brain promotes glial activation and accelerates tau pathology in a mouse model of tauopathy. Front Immunol 2020; 11: 997. 8 Wu T, Dejanovic B, Gandham VD, et al. Complement C3 is activated in human AD brain and is required for neurodegeneration in mouse models of amyloidosis and tauopathy. Cell Rep 2019; 28: 2111–23.9 Gratuze M, Schlachetzki JCM, D’Oliveira Albanus R, et al. TREM2-independent microgliosis promotes tau-mediated neurodegeneration in the presence of ApoE4. Neuron 2023; 111: 202–19. 10 Bussian TJ, Aziz A, Meyer CF, Swenson BL, van Deursen JM, Baker DJ. Clearance of senescent glial cells prevents tau-dependent pathology and cognitive decline. Nature 2018; 562: 578–82. 11 Paolicelli RC, Sierra A, Stevens B, et al. Microglia states and nomenclature: a field at its crossroads. Neuron 2022; 110: 3458–83. 12 Kitazawa M, Oddo S, Yamasaki TR, Green KN, LaFerla FM. Lipopolysaccharide-induced inflammation exacerbates tau pathology by a cyclin-dependent kinase 5-mediated pathway in a transgenic model of Alzheimer’s disease. J Neurosci 2005; 25: 8843–53. 13 Bhaskar K, Konerth M, Kokiko-Cochran ON, Cardona A, Ransohoff RM, Lamb BT. Regulation of tau pathology by the microglial fractalkine receptor. Neuron 2010; 68: 19–31. 14 Krstic D, Madhusudan A, Doehner J, et al. Systemic immune challenges trigger and drive Alzheimer-like neuropathology in mice.