Increased release of immunoreactive cholecystokinin octapeptide by morphine and potentiation of mu-opioid analgesia by CCKB receptor antagonist L-365,260 in rat spinal cord.

Increased release of immunoreactive cholecystokinin octapeptide by morphine and potentiation of mu-opioid analgesia by CCKB receptor antagonist L-365,260 in rat spinal cord.
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在大鼠脊髓中吗啡增加了免疫反应性胆囊收缩素八肽的释放,CCKB 受体拮抗剂 L-365,260 增强了 mu-阿片类镇痛作用。

DOI:
10.1016/0014-2999(93)90948-h
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发表时间:
1993
影响因子:
5
通讯作者:
Han,JS
Han,JS
中科院分区:
医学2区
文献类型:
--
作者:
Zhou,Y;Sun,YH;Zhang,ZW;Han,JS

文献摘要

被引文献

相似文献

本文首次报道了在体研究中,全身注射吗啡可使大鼠脊髓灌流液中八肽胆囊收缩素(CCK-8)的免疫反应性显著增加(89%,P < 0.01),而纳洛酮可完全逆转这种作用。由于CCK-8已被证明在脊髓水平具有有效的抗阿片活性,因此预期脊髓胆囊收缩素效应的阻断将增强阿片镇痛。以甩尾潜伏期为伤害性指标,发现鞘内(i.t.)腹腔注射新型CCK-8拮抗剂L-365,260可明显增强μ阿片受体激动剂吗啡(4 mg/kg s.c.)或羟甲芬太尼(32 ng i.t.)。CCKA拮抗剂devazepide在剂量比L-365,260高40-50倍时也获得了类似的效果。地伐卓和L-365,260均显示钟形剂量-反应曲线。结果证实了CCK-8释放增加可能构成脊髓水平阿片效应的自限性过程,CCK-8受体介导了CCK-8在脊髓的抗阿片效应。
This is the first report showing, in an in vivo study, that systematic morphine produced a marked (89%, P < 0.01) increase of the cholecystokinin octapeptide (CCK-8) immunoreactivity in the perfusate of the rat spinal cord, an effect completely reversed by naloxone. Since CCK-8 has been shown to possess potent anti-opioid activitit at a spinal level, a blockade of the spinal cholecystokinin effect would be expected to potentiate opiate analgesia. With tail flick latency as a nociceptive index, it was found that intrathecal (i.t.) injection of a novel CCKBantagonist L-365,260 produced a marked potentiation of the analgesic effect induced by the μ-opioid agonists morphine (4 mg/kg s.c.) or ohmefentanyl (32 ng i.t.). Similar effects were obtained with the CCKAantagonist devazepide at a dose 40–50 times higher than that of L-365,260. Both devazepide and L-365,260 showed a bell-shaped dose-response curve. The results confirm the notion that an increased release of CCK-8 may constitute a self-limiting process for opioid effects at the spinal level, and that it is the CCKBreceptor which mediates the anti-opioid effect of CCK-8 in the rat spinal cord.