HEART RATE DURING CONDITIONING IN HUMANS: EFFECTS OF UCS INTENSITY, VAGAL BLOCKADE, AND ADRENERGIC BLOCK OF VASOMOTOR ACTIVITY.
HEART RATE DURING CONDITIONING IN HUMANS: EFFECTS OF UCS INTENSITY, VAGAL BLOCKADE, AND ADRENERGIC BLOCK OF VASOMOTOR ACTIVITY.
复制标题
人体调节期间的心率：UCS 强度、迷走神经阻滞和血管舒缩活动的肾上腺素能阻滞的影响。
DOI：
10.1037/h0022033
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发表时间：
1965
影响因子：
--
通讯作者：
M. Pérez
中科院分区：
文献类型：
--
作者：
P. Obrist;D. M. Wood;M. Pérez
在经典条件反射实验中，研究了心脏迷走神经和交感神经支配以及外周反射机制对心率减慢的影响。研究发现，迷走神经约束的增加是减慢反应的基础，而减慢反应反过来又掩盖了交感加速效应的表现。后者仅在迷走神经被药物阻断和使用非常强烈的UCS时观察到。当条件加压反应在药理学上被阻断时，通过直接记录动脉血压来评估，减速反应并没有改变，这表明外周稳态反射机制不是这种反应的基础，它很可能是一种条件反应。（25 ref.）（PsycINFO Database Record (c) 2016 APA，版权所有）
2 experiments concerned with the influence of the vagal and sympathetic innervations of the heart and of peripheral reflex mechanisms are reported on the deceleration of heart rate observed on test trials during classical conditioning in human Ss. An increase in vagal restraint was observed to be the basis of the deceleratory response, which in turn masks the manifestation of sympathetic acceleratory effects. The latter were only observed when the vagus was pharmacologically blocked and when a very intense UCS was used. When the conditioned pressor responses were blocked pharmacologically, as evaluated by direct recordings of arterial blood pressure, the deceleratory response was not changed, indicating that the peripheral homeostatic reflex mechanisms are not the basis for this response and that it is likely a conditioned response.(25 ref.)(PsycINFO Database Record (c) 2016 APA, all rights reserved)