Loss of DEP-1 (Ptprj) promotes myeloproliferative disease in FLT3-ITD acute myeloid leukemia

Loss of DEP-1 (Ptprj) promotes myeloproliferative disease in FLT3-ITD acute myeloid leukemia
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DOI：
10.3324/haematol.2017.185306
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发表时间：
2018-06
期刊：
Haematologica
影响因子：
10.1
通讯作者：
A. Kresinsky;R. Bauer;T. Schnöder;T. Berg;D. Meyer;Volker Ast;R. König;H. Serve;F. Heidel;F. Böhmer;Jörg P Müller
A. Kresinsky;R. Bauer;T. Schnöder;T. Berg;D. Meyer;Volker Ast;R. König;H. Serve;F. Heidel;F. Böhmer;Jörg P Müller
中科院分区：
医学1区
文献类型：
--
作者：
A. Kresinsky;R. Bauer;T. Schnöder;T. Berg;D. Meyer;Volker Ast;R. König;H. Serve;F. Heidel;F. Böhmer;Jörg P Müller

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Acute myeloid leukemia (AML) is a heterogeneous group of diseases caused by oncogenic transformation of hematopoietic stem and progenitor cells. Mutations in the gene encoding the tyrosine kinase FLT3 leading to internal tandem duplications (ITDs) of sequence represent one of the most frequent