Long-lasting reductions of spasticity induced by skin electrical stimulation.
Long-lasting reductions of spasticity induced by skin electrical stimulation.
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DOI：
10.1109/86.547923
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发表时间：
1996-12-01
期刊：
影响因子：
--
通讯作者：
Rymer, W Z
中科院分区：
文献类型：
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作者：
Dewald, J P;Given, J D;Rymer, W Z
我们研究了电刺激皮肤对9名偏瘫中风患者上肢痉挛的影响。通过比较在低强度皮肤刺激之前和之后记录手肘的斜坡和保持角度扰动期间引起的反射扭矩反应来量化效果。以20 Hz频率、0.1 ms脉冲持续时间、低于运动阈值但高于感觉阈值的强度水平对二头肌肌肉上的皮肤施加电刺激10分钟。在7的9名受试者，刺激皮肤痉挛肌肉减少峰值扭矩反应在屈肌和伸肌至少30分钟。在这7名受试者中有显着增加的平均阈值角反射扭矩的发病，使更大的角度旋转需要启动牵张反射反应。这种变化发生时反射阻抗没有变化。这些反射力矩的长期变化的起源尚不清楚，但可能反映了牵张反射环路外脊髓回路的突触可塑性。
We studied the effects of electrical stimulation of the skin on upper extremity spasticity in nine hemiparetic stroke subjects. The effects were quantified by comparing reflex torque responses elicited during ramp and hold angular perturbations of the elbow recorded before and after low-intensity skin stimulation. Electrical stimulation was applied to skin over the biceps muscle for a period of ten minutes at a 20 Hz frequency, pulse duration 0.1 ms, with an intensity level below motor threshold but above sensory threshold. In seven of the nine subjects, stimulation of skin over spastic muscle reduced peak torque responses in both flexors and extensors for at least 30 min. In these seven subjects there were significant increases in mean threshold angle for the onset of reflex torque so that a greater angular rotation was required to initiate the stretch reflex response. This shift occurred without change in reflex impedance. The origins of these long-term changes in reflex torque are unclear, but may reflect synaptic plasticity of spinal circuitry outside the stretch reflex loop.