Electron donor dependent inhibition mechanisms of D-phenylalanine on corrosion of Q235 carbon steel caused by Desulfovibrio sp. Huiquan2017

Electron donor dependent inhibition mechanisms of D-phenylalanine on corrosion of Q235 carbon steel caused by Desulfovibrio sp. Huiquan2017
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D-苯丙氨酸对脱硫弧菌引起的Q235碳钢腐蚀的电子供体依赖性抑制机制。

DOI:
10.1016/j.corsci.2021.109493
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发表时间:
2021
期刊:
影响因子:
8.3
通讯作者:
Zhu Liyang
Zhu Liyang
中科院分区:
材料科学1区
文献类型:
--
作者:
Li Ee;Wu Jiajia;Zhang Dun;Wang Peng;Wang Yu;Xu Ming;Li Ce;Sun Zhihua;Zhu Liyang

文献摘要

相似文献

D-苯丙氨酸对Q235碳钢硫化弧菌腐蚀的作用机理汇泉2017与不同的电子供体进行了调查。当乳酸为主要电子供体时,D-Phe不影响平均腐蚀损失,但通过抑制生物膜的形成和改变生物膜结构来抑制局部腐蚀,这一点得到了比较转录组分析的支持。去除乳酸和酵母膏后,D-Phe显著减少了腐蚀损失,减弱了选择性腐蚀和局部腐蚀。这可能是由于Stickland反应的进行所致,其中L-半胱氨酸是电子供体,D-苯丙氨酸被细菌转化为L-苯丙氨酸是电子受体。
Mechanisms of D-phenylalanine (D-Phe) on corrosion of Q235 carbon steel caused byDesulfovibriosp. Huiquan2017 with different electron donors were investigated. When lactate was main electron donor, D-Phe did not influence the average corrosion loss, but inhibited localized corrosion by repressing biofilm formation and altering biofilm structure, which was supported by comparative transcriptome analysis. When lactate and yeast extract were removed, D-Phe significantly reduced corrosion loss, and weakened selective phase corrosion and localized corrosion. It may due to the proceeding of the Stickland reaction, in which L-cysteine is electron donor and L-Phe transformed from D-Phe by bacteria is electron acceptor.