Superoxide dismutase expression attenuates cigarette smoke- or elastase-generated emphysema in mice

Superoxide dismutase expression attenuates cigarette smoke- or elastase-generated emphysema in mice
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DOI:
10.1164/rccm.200506-850oc
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发表时间:
2006-03-15
影响因子:
24.7
通讯作者:
D'Armiento, JM
D'Armiento, JM
中科院分区:
医学1区
文献类型:
--
作者:
Foronjy, RF;Mirochnitchenko, O;D'Armiento, JM

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理由:氧化剂被认为在肺气肿的形成中起着重要作用。目的:探讨人铜锌超氧化物歧化酶(CuZnSOD)在小鼠肺内的表达是否对肺气肿的发生有保护作用。方法:将转基因CuZnSOD小鼠和同窝小鼠分别暴露于香烟烟雾中(6 h/d, 5 d/周,持续1年),并与未暴露的小鼠进行比较。第二组采用气管内弹性蛋白酶诱导肺气肿。测量方法:通过细胞计数和髓过氧化物酶水平测量肺部炎症。采用免疫荧光法检测3-硝基酪氨酸和8-羟基脱氧鸟苷的氧化损伤及脂质过氧化水平。通过测量平均线性截距(L-m)来判断肺气肿的发展。主要结果:吸烟引起嗜中性粒细胞炎症增加4倍,肺髓过氧化物酶活性增加1倍。这种炎症反应在暴露于烟雾中的CuZnSOD小鼠中没有发生。同样,CuZnSOD的表达阻止了烟雾暴露后肺脂质过氧化产物增加58%。最重要的是,CuZnSOD在烟雾诱导模型(L-m, 68暴露对照与58暴露转基因对照,p < 0.04)和弹性酶生成模型(L-m, 80暴露对照与63暴露转基因对照,p < 0.03)中均能预防肺气肿的发生。这些结果首次证明,抗氧化剂可以预防烟雾引起的炎症,并可以抵消导致肺气肿形成的蛋白水解级联反应,在两种不同的动物模型中。结论:这些发现表明,旨在增强或补充肺部抗氧化剂的策略可能有效地预防和治疗该疾病。
Rationale: Oxidants are believed to play a major role in the development of emphysema.Objectives:This study aimed to determine if the expression of human copper-zinc superoxide dismutase (CuZnSOD) within the lungs of mice protects against the development of emphysema.Methods: Transgenic CuZnSOD and littermate mice were exposed to cigarette smoke (6 h/d, 5 d/wk, for 1 yr) and compared with nonexposed mice. A second group was treated with intratracheal elastase to induce emphysema. Measurements: Lung inflammation was measured by cell counts and myeloperoxidase levels. Oxidative damage was assessed by immunofluorescence for 3-nitrotyrosine and 8-hydroxydeoxyguanosine and lipid peroxidation levels. The development of emphysema was determined by measuring the mean linear intercept (L-m).Main Results: Smoke exposure caused a fourfold increase in neutrophilic inflammation and doubled lung myeloperoxidase activity. This inflammatory response did not occur in the smoke-exposed CuZnSOD mice. Similarly, CuZnSOD expression prevented the 58% increase in lung lipid peroxidation products that occurred after smoke exposure. Most important, CuZnSOD prevented the onset of emphysema in both the smoke-induced model (L-m, 68 exposed control vs. 58 exposed transgenic; p < 0.04) and elastase-generated model (L-m, 80 exposed control vs. 63 exposed transgenic; p < 0.03). These results demonstrate for the first time that antioxidants can prevent smoke-induced inflammation and can counteract the proteolytic cascade that leads to emphysema formation in two separate animal models of the disease.Conclusions: These findings indicate that strategies aimed at enhancing or supplementing lung antioxidants could be effective for the prevention and treatment of this disease.